The 52-year-old American man was diagnosed with neurocysticercosis. His only habit connected to the parasitic infection was eating "lightly cooked, non-crispy bacon," new study says.

A man was hospitalized with worsening migraines only to find out they were caused by parasitic tapeworm larvae in his brain — and researchers believe he was infected by eating undercooked bacon.

The unidentified 52-year-old American man consulted doctors about changes in his usual migraines over four months, according to a study in the American Journal of Case Reports published Thursday. The migraines became more frequent, severe and unresponsive to medication.

The patient was admitted to the hospital for testing. CT scans revealed numerous cystic foci, which are fluid-filled sacs in the brain. Cysticercosis cyst antibody tests returned positive, and the man was diagnosed with neurocysticercosis, the study said.

  • evasive_chimpanzee@lemmy.world
    49·
    8 months ago

    I posted this in a reply, but I’m reposting it here:

    Bacon is already cooked when you buy it from the store (at least in america). The difference between “pork belly” and “bacon” is that bacon is cured and then smoked. That smoking process gets the temperature above the range to cook it.

    Besides, even “chewy” bacon is going to get above the recommended pork cooking temp. It’s so thin, it will reach that temperature very quickly. If it gets hot enough to start rendering the fat, it’s definitely hot enough, and I have a hard time believing anyone would pull bacon out of the oven before it’s had time to render some fat.

    If you read the case report by the docs, they don’t say “we think he got this disease from bacon”.

    Our patient’s lifelong preference for soft bacon may have led to instances of undercooked bacon consumption, but this would have caused him to develop taeniasis, an intestinal tapeworm, and not cysticercosis [1]. Taeniasis occurs when consuming undercooked pork and the larval cysts embedded within, while cysticercosis is contracted when humans ingest eggs found in the feces of other humans with taeniasis [1].

    In other words, the brain version only comes from eating the eggs, which can only come from eating poop of someone who had the intestinal worms. They later specify that they think the most likely scenario is that he got the intestinal version, re-infected himself through the fecal-oral pathway, then somehow cured himself of the intestinal version.

    Personally, I think the most likely scenario is that he got the parasite the same way you get any other disease that uses the fecal-oral pathway: contact with someone’s inadequately washed hands, or eating food prepared by someone with inadequately washed hands.